Dr. Gallagher has had a long-standing defined clinical and translational interest in the mechanisms underlying impaired diabetic wound healing. Her long-term goal is to uncover the basic mechanisms responsible for the coordination of immune cells in early tissue repair processes, determine impairments in Type 2 Diabetes and translate this knowledge to the bedside. It is my belief that the diabetic phenotype has systemic effects on bone marrow cells that alter innate immune cell phenotypes resulting in impaired peripheral wound healing.

Since joining the faculty at the University of Michigan, Dr. Gallagher's research has focused on histone methylation changes in bone marrow progenitor cells and the impact on peripheral macrophage phenotypes as it relates to wound healing and inflammation. This work will provide important data on inflammatory macrophages in peripheral tissue and their contribution to chronic inflammation and may lead to novel treatment modalities. I have also trained numerous post-doctoral fellows/residents in my lab under NIH T32, F32, AAS grant, VESS grant and other funding mechanisms. I have a strong interest in training postdocs/residents in translational science.